Showing posts with label back pain atlanta. Show all posts
Showing posts with label back pain atlanta. Show all posts

Wednesday, March 20, 2013

Biomechanical Issues for Motor Vehicle Collisions



Biomechanical Issues for Motor Vehicle Collisions

It is important for the doctor to recognize that if a particular case goes into litigation, the insurance carrier will often hire a defense biomechanics expert, or medical expert that uses the conclusions of the biomechanists, to challenge the patient’s injuries and symptom logy, as well as the doctor’s diagnosis and treatment. The biomechanist can be an effective ally for the defense if he/she can attack the injury mechanisms and G forces (acceleration) generated during the collision, and influence a jury to believe the patient was not injured or simply had a trivial injury that would have resolved within a brief period of time with or without any treatment. These opinions are then typically reiterated by the defense medical expert, who is in agreement with the biomechanist, with the clear objective of downplaying the extent of the plaintiff’s injuries (bias) and attacking the treating doctors. In most cases the defense biomechanics expert will focus on the following issues:
               -Insufficient force to cause injury
               -Downplay the severity of injury, this giving the defense doctor basis to state that the treatment was excessive and unnecessary
               -Forces of collision are similar to those experienced in activities of daily living and therefore not responsible for post crash symptoms or injuries
               -Will often rely on limited living human volunteer testing (no subjects taken from general population) and extrapolate data to the specific patient
               -No injury mechanism
               -All post-traumatic symptoms were due to prior degenerative conditions or injuries and would have occurred at the same time frame absent the MVC

Monday, March 11, 2013

Distraction Test



Distraction Test

The complaints of patients with chronic or degenerative conditions of the cervical disc are quite different from those of patients with acute conditions. Patients with chronic conditions experience intermittent episodes of pain, discomfort, and muscle spasm. Exacerbations come from exertion. Pain and stiffness may result from weather changes or unexplained causes. Radiculopathy is not always present. Hyporeflexia, motor weakness, and sensory disturbance (especially paresthesia) are common.

1.  Delay in the onset of symptoms.  If the symptoms are not written down and documented within a few hours or days after the crash, then it is very difficult for the patient to say he/she had pain when there is no written evidence.  This can be a critical issue in a case, for example, the first doctor to note that the patient had a traumatic brain injury was made 8 to 10 months after the collision.

2.  Delay in seeing the first doctor.  Any significant delays between the date of the crash and seeing the doctor may create credibility issues for the case.  The patient needs to have a good explanation for waiting for several days to weeks for the first appointment.  There may be legitimate issues such as, some doctors have very busy schedules and may not have appointments available for several days.  The reasons for delays should be noted in the file.  The doctor or patient may have a vacation or work trip scheduled, be out of town for a medical emergency, may have an unrelated surgery that made it impossible to make an appointment, or may simply have not transportation available.

3.  There were conflicts in the history given by the patient in the records.  For example, patient stated in his deposition that he had never been involved in a crash before.  Records from 1989 indicate that the patient was involved in a rear-end crash.  The ER doctor notes that the patient did not use a seatbelt and the orthopedist noted that the patient did use a seatbelt.

4.  Little-to-no damage to vehicle.  The defense will advocate that the damage to the vehicle equals the injury potential (see Chapter 15 for crash speed thresholds for injuries).  There are generally few photographs taken of the vehicles that adequately show the extent of the damage.  Most jurors are going to see poor quality blow-ups of the vehicles or as the parts are removed for repairs.

5.  Impact forces not sufficient to cause any injury or was sufficient enough to have caused only mild muscular strain that would have healed without treatment within a few days may have occurred.  No mechanism of injury was possible in this collision.

6.  No objective findings to prove injuries.  No bruises, bleeding, lacerations, fractures, or photographic evidence of the injury to show the jury.

7.  The treatment that was provided was passive and has not been proven to work.  Patient’s condition would have been the same with or without treatment.  Exercise is the only thing that works.  Patient had only palliative benefits from the treatment.  No long-term benefits noted in file.  May look at deposition.  If patient states in the deposition that the treatment only helped for one to two weeks but the patient continued to have treatment for an additional four months. The case may have challenges.

8.  The duration of treatment was too long, was excessive, or was duplicative, and therefore is not justified from doctor’s experience.  The treatment costs were thus unreasonable for the mild nature of the injuries.  The osteopath, chiropractor, and physical therapist were doing similar things, and therefore the treatments were unnecessary.

9.  Gaps in treatment indicate that the patient did not have any pain.  That a reasonable person in pain would see a doctor is a common attack.

10.  Healing and full recovery takes two to four weeks.  This opinion is simply a hoax for most cases (see Chapter 7 for more about soft tissue healing).

11.  Every person will have full recovery following whiplash injuries.  Doctors and attorneys are to blame.  This is another hoax (see Chapter 13 for a review of prognostic studies).

12.  Patient saw too many providers, consistently self-referred himself or herself, and had a history of psychological problems.

13.  Documentation was poor.  The doctors did not note symptoms, or there are inconsistent statements made by the doctors.

14.  New injuries, including MVCs, falls, etc., or simply flared-up bending over are responsible for the problems.

15.  Prior injuries resulted in all of the problems.  The defense may attack the case by telling the jury that the injuries sustained 8 to 15 years earlier were responsible for the current pain, although there is not evidence of the patient being seen by any health providers for the past five to six years for any musculoskeletal pain.

16.  Prior pain and / or treatment for this pain within the past couple years clearly indicates that the patient’s pain was long-term and would have been present despite the crash.

17.  No justification for the amount of time off work.  May state that the typical patient is back to work within one week (see Chapter 12 for more information).

18.  The MRI scan results showing a bulging or herniated disc are also seen in the general population and are not related to the accident.  Typically, if any degeneration or spurring is noted in the radiology report, the defense medical and biomechanics expert will use that as his/her basis for that opinion using a “Natural Progression” theory.

19.  Future treatment is not needed.  The defense attorney may tell the jury in the opening statement that “Everyone knows that once the case settles the plaintiff’s pain will go away.”

20.  Conservative jurors who have a “Hollywood mentality” can make a case extremely difficult even with the best documentation, proof of bulging discs, and great doctors.  The jury may perceive the plaintiff as looking healthy and there are no photographs of blood and guts.  Some court districts are known as being very conservative, thus making it difficult to get any significant awards.  Some jurors may see that there is monetary motivation for the plaintiff.

Wednesday, March 6, 2013

Brachial Plexus Tension Test


Brachial Plexus Tension Test

Clinical Pearl

Although the brachial plexus tension test involves shoulder joint movement, it also provides maximum stretch on the brachial plexus, which affects the lower branches of the cervical spine (C5) the most. If this test is positive, the early stages of a C5 nerve root disorder may be present along with the subtle signs of a positive doorbell sign (pain that occurs at the superior scapulovertebral border and radiates with the use of deep palpation of the C5 segment) and pain in the deltoid area. The deltoid pain is often misconstrued as an articular problem of the shoulder.


Dejerine’s Sign

Clinical Pearl

Patients with radicular symptoms and pronounced Dejerine’s sign, especially if it is in the lumbar spine, should be told to bend the knees and lean into a wall during a cough or sneeze. This maneuver reduces intradiscal pressure and minimizes the effect of the cough or sneeze on the nerve root. A more worrisome situation is the sudden, unexpected absence of Dejerine’s sign when all other clinical findings indicate an active nerve root compression. The loss of the sign indicates fragmentation of the disc with momentary decompression of the nerve.

Saturday, February 23, 2013

Disability and Handicap


Disability and Handicap

Disability is a present when a tissue, organ, or system cannot function adequately. A handicap exists when disability interferes with a patient’s daily activities or social/occupational performance. A marked disability does not necessarily cause a handicap. Conversely, minor disability may produce a major handicap. Both conditions require separate assessment. Patients’ perception of their problems will be molded by their adaptation to the depreciated tissue as well as their aspirations for recovery.

Assessing Disability

An aid in assessing the more important aspects of disability is the PILS mnemonic, which considers four issues:

1.       P Preventable causes of disability (e.g., falls, direct trauma)
2.      I Independence (e.g., self-care)
3.      L Lifestyle (roles, goals)
4.      S Social factors (e.g., family, friends, shelter)


Functional Assessment

A complete functional assessment includes evaluation of the following:
1.      Self-care: ability to wash, bath, attend to toilet needs, dress, cook, and feed oneself
2.      Mobility: ability to stand, transfer, walk, negotiate stairs, drive, and use public transportation
3.      Lifestyle: nature of occupation, work capacity, and Social Security benefits

Monday, February 18, 2013

CRASH-RELATED FACTORS


CRASH-RELATED FACTORS

Numerous crash-related factors have been investigated for their predictive capacity. Some of these include the vehicle being stationary when hit, a frontal collision, a rear-end collision, side or other collision, being unprepared for the collision, no seat belt use and no head restraint. However, few of these factors have demonstrated significant predictive power. The exceptions to this are not wearing a seat belt, which was reported to nearly double the risk of developing persistent pain or disability. This is an interesting finding as, in certain jurisdictions where compulsory seat belt use is legislated, the voluntary admission of not wearing a seat belt would not be expected to be common, particularly in jurisdictions operating under a fault-based system. Thus, it is possible that the risk of developing a chronic condition associated with not wearing a seat belt may be even higher than that reported by Walton and colleagues. Scholten-Peeters et al. reported some limited prognostic value for accidents that occur on a highway, but also reported that there was strong evidence that rear-end collisions have no predictive value for poor functional recovery.
Thus, the weight of evidence indicates that crash-related factors are of limited value in attempting to predict poor recovery following whiplash injury.


SOCIODEMOGRAPHIC FACTORS

The predictive capacity of sociodemographic factors has been studied extensively in musculoskeletal pain conditions, and whiplash is no exception. Older age (>50 years), while showing predictive 

Tuesday, February 12, 2013

PROGNOSTIC FACTORS FOR NON-RECOVERY



PROGNOSTIC FACTORS FOR NON-RECOVERY


            The capacity to predict outcome following whiplash injury is important for several reasons. Predictive factors may be modifiable or non-modifiable, and treatments directed at the former factors may improve outcomes for those identified as at risk of poor recovery. This, in turn, may assist in the curtailment of both personal and financial costs associated with the condition. The identification of those who show good potential for recovery is also important so that both injured people and clinicians can have greater confidence in a good outcome. An understanding of prognostic indicators for both outcomes will allow the appropriate allocation of resources by policy-makers.
Fifteen years ago, the Quebec Task Force identified predictive studies as an area requiring urgent investigation in whiplash research. Since that time the number of cohort studies has substantially increased and now several systematic reviews of prognosis are available. However, these have not been undertaken without difficulty owing to shortcomings in some of the primary cohort studies, including inconsistencies between studies in time from injury until baseline data collection, time to follow-up and use of various and sometimes un-validated outcome measures. There is also variation between the systematic reviews, with some performing meta-analysis, others indicating that due to heterogeneity data pooling could not be undertaken, and others comprising task force findings that were not peer reviewed. Nevertheless, the findings of the various reviews have generally been in agreement that the factors of higher initial predictors of poor functional recovery.
It is not the aim of this chapter to conduct another systematic review of currently available cohort studies. Rather, it is to present an overview of systematic potential findings, discuss emerging factors that show potential for prognostic capacity of both recovery and non-recovery, and outline the clinical implications for the recognition of prognostic indicators.


PRESENTING SIGNS AND SYMPTOMS

Clearly, the most consistent predictor of poor functional recovery is the intensity of neck pain at the initial or baseline assessment point. Walton et al. synthesized the data from eight cohorts and established a cut-off point of 55 out of 100 or 5 out of 10 on a visual analogue pain scale. These authors report that a pain intensity of greater than 55 out of 100 demonstrated a nearly six-fold (OR, 5.77; 95% CI: 2.89-11.52) increase in the risk of persistent pain or disability at follow-up. This factor was slightly more robust at predicting an outcome of disability when compared to pain outcomes. Initially, moderate-to-high levels of pain-related disability have also shown predictive capacity.
Some reviews pointed to other symptoms, such as the presence of headache, or neurological symptoms, such as arm pain or paresthesia, as also showing predictive capacity. The Quebec Task Force classification (see Chapter 1 for description), a predominantly symptom-based system, was evaluated in two reviews, with the authors concluding that increasing grades of whiplash associated disorders (WAD) predicted increasingly higher pain intensities and disability two years later. Walton et al. reported that the size of effect was significant when WAD grades II and III were compared against grades I and 0, and this effect was consistent at various follow-up times points.
Other symptoms, such as dizziness, reported sleep disturbances and cognitive difficulties, have not emerged from the systematic reviews as showing any predictive capacity.

Saturday, February 2, 2013

AN EPIDERMIOLOGICAL APPROACH TO AETIOLOGY


AN EPIDERMIOLOGICAL APPROACH TO AETIOLOGY

In epidemiological, one of the aims is to assess the independent association between a potential risk or prognostic factor. It is, however, extremely important to keep in mind that in most disease and injuries, a multifactorial casual model is needed to understand the onset and prognosis.
This can be effectively discussed using the ‘pie’ model, or the component cause model, introduced to epidemiologists by Rothman. Some prognostic factors that may be involved in recovery from WAD are introduced in two different ‘pie’ models.

According to the ‘pie’ model, all contributing causes in one of the sufficient causes, or ‘pie’ are needed, in order to recover from WAD. Each sufficient cause represents one of presumably several alternative routes leading to recovery. The interactions between contributing causes are illustrated as separate ‘pie slices’ included in the same pie. Slices in one ‘pie’ are said to interact, because it is their joint action that leads to recovery. In real life, of course, different distributions of biological, psychological and social factors occur among different people.

In the literature, there is no recognized necessary for recovery from WAD. Neither has any sufficient cause been identified. Instead, several contributing causes (i.e. factors that affect the cause of recovery) have been found. When linking this ‘pie’ model to the evidence of prognostic factors in WAD, numerous sets of possible ‘individual pies’ become obvious. In summary, several different sufficient causes for recovery from WAD exist; in other words, several alternative routes lead to recovery.

In research, the effect of individuals contributing causes, or risk factors, are usually investigated for the condition in question. Complete sufficient causes usually do not lend themselves to studies because few of these scenarios actually exist (exceptions include bacterial infection). Still, it is important to keep the ‘pie’ model in mind when interpreting and discussing empirical results on risk and prognosis. A single contributing cause may otherwise be confused for a sufficient cause. The whole picture for specific sets of sufficient cause is seldom discussed in the research of WAD or in other contexts.

The casual process is more complicated than explained by the ‘pie’ model, since effects are more likely to depend on other factors that happen simultaneously. For instance, some persons in pain may call for support from family members or workmates, which in turn may change the person’s future pain perception and behavior (positively or negatively). In other instances, pain intensity may cause depressed mood, or depressed mood may cause more pain (bidirectional). Nevertheless, this component cause model is a fruitful way to conceptualize WAD as it visualizes the multifactorial nature of WAD and how biological, psychological and social factors may interact.

Wednesday, January 23, 2013

CUMULATIVE INCIDENCE OF AND RISK FACTORS FOR WAD


CUMULATIVE INCIDENCE OF AND RISK FACTORS FOR WAD



The cumulative incidence is the number of new cases of an event or outcome occurring in a population over a certain time period. Some evidence from the literature indicates that the incidence of WAD differs between countries. There is also some evidence that the incidence of WAD has increased from the beginning of the 1990s to after the year 2000, with the annual incidence for the latter period being about 300 per 100,000 inhabitants in the studies where emergency setting visits are used. In some instances, the increase is between three and tenfold. It is not known if this increase is partly due to a change in care-seeking behavior.
There are also some indications from administrative insurance claims database in different European countries (e.g. Norway, the Netherlands and Sweden) of a reduction in the number of WAD claims, whereas such decreases have not been seen in Denmark or the United Kingdom. Sweden, for instance, has seen a 33% decrease in personal motor vehicle crash (MVC) injuryclaims between 2002 and 2008. The relative decrease is similar between the incidence of WAD and other types of injuries, with WAD constituting about 50% of all MVC injury claims. This decrease is not due to reduction in the number of MVCs, and nor has the insurance system in Sweden changed. Instead, this decrease is likely to be due to a combination of reasons. For example, some care manufacturers have developed whiplash-protection devices for new car models, which presumably will result in fewer cases of WAS as a result of rear-end collisions. Secondly, during the second half of the 1990s, police personnel in Sweden showed an increased awareness that there is no need to advise car occupants to seek healthcare if no symptoms are present. Thirdly, the mass media focus in Sweden on whiplash has decreased substantially from over 800 articles in the beginning of the 2000s to only about 200 articles in 2008.
Incidence calculation through insurance claims may be prone to other forms of bias. For instance, insurance systems where there are no benefits for the person responsible for a collision may underestimate the frequency of injuries, since fewer claims would be reported. This would also happen with insurance systems where insurance claim access us limited, or where payments for compensation result in a significant increase in the insurance premium. On the other hand, healthcare data may also be prone to bias, since such data only captures those who seek the type of healthcare utilization in question (e.g. emergency care).

Sunday, January 13, 2013


Crash Speeds and Injury Risk: Epidemiologic and Forensic Considerations





Crash-related injuries are the most frequently litigated type of injury in the United States, primarily due to two facts: motor vehicle crashes are one of the most common sources of injuries in society, and many crash-related injuries result from negligence of another individual. Litigation naturally produces a polar alignment of opinion, with one side favoring the plaintiff and the other side favoring the defense. There is no question as to which side has the greatest economic leverage, as auto insurers are multibillion dollar corporations with the added advantage of legislatively-mandated insurance coverage for any vehicle being operated on a public roadway. Thus, if an individual is legally operating a motor vehicle in the United States, that individual almost certainly is paying premium dollars to an insurance company.
               For many years, insurers have invested premium income in a variety of financial instruments and in times of positive economic growth this has proven an effective strategy for increasing their income. In times of stagnant or negative economic growth, insurers must look for other ways to increase profits to satisfy cost-of-living increases for their employees, as well as continuing profits for their owners and investors. This has led to a more recent method, which is to aggressively reduce payments on claims. One of the most effective methods for reducing such payments is to deny payment based on an allegation that a claim is fraudulent or “built up.” No one would argue with the premise that an insurer has the right to protect itself from fraudulent claims, and insurers have capitalized on this right by expanding their definition of what they consider as a fraudulent or built up claim, definitions that are not necessarily validated with data indicating that such claims are necessarily specious.
               A prime example of this expanded definition is the minor impact soft tissue (MIST) claims management protocol initiated by Allstate Insurance in the mid-1900s and adopted by virtually every other US insurer to one extent or another. The premise of this program is that the amount of vehicle damage is an indicator of whether or not a crash could have or should have resulted in an injury. Thus, according to claims adjuster training, an occupant who claims an injury with no more than bumper damage to his or her vehicle is automatically considered to be a likely fraud, despite the presence of medical documentation of injury. The result of this strategy is that, without any valid scientific medical documentation to support their position, the insurance industry has drawn an arbitrary line as to the type of injury claims they will pay versus those they won’t. The real criterion is more likely based in the types of cases insurers believe they are most likely to win in front of a jury: those in which an injury claim can be made to appear counterintuitive or contrary to the “common sense” of a layperson.
               The defense of the litigation of such cases has generated an entire cottage industry in and of itself, with “biomechanists” and “biomechanical engineers” (increasingly referring to individuals with little or no training in these fields) claiming that injuries cannot occur in minimal damage crashes because experimental studies of human volunteer crashes and cadaver, dummy, and animal experiments have somehow proven otherwise. The basis of the MIST defense is the precept that there is a minimum force threshold below which no injury is likely to occur, and that the force threshold coincides with a round money figure in vehicle damage (typically $1000 or less) regardless of the vehicle. Recent trades in litigation practices have illustrated the fact that minimal damage crashes are merely a beachhead in injury threshold defense practices. An increasing practice for many defense experts is to claim that no injury is possible in minimal damage crashes and that no permanent injury is possible in larger crashes without fracture or dislocation.
               Experts who regularly work for the defense in crash injury litigation must rely on an insistence that there is a legitimate scientific basis from which to claim that a crash injury threshold below which injury cannot or is unlikely to occur exists for certain crashes. This premise is fallacious, as numerous epidemiologic studies demonstrate that a variety of injuries can and do occur in a variety of crashes. However, many experts obfuscate this fact with references to government standards and research that refers to injury thresholds. The use of technical jargon to obscure a complete lack of valid basis underlying opinion is called “junk science,” and courts regularly exclude such testimony once it is revealed as such. The purpose of this chapter is to help the reader understand the foundations for references to “injury thresholds,” how they are developed, and why they are not applicable to victims of real-world crash-related trauma.
               While the information in this chapter was derived from a variety of sources, the majority of data regarding real-world crashes have been published by the National Highway Traffic Safety Administration (NHTSA) from the data collection efforts represented in their National Automotive Sampling System (NASS). Additionally, NHTSA collects data on vehicle crashworthiness with their frontal and side barrier tests. The literature cited in the chapter comes from the Society of Automobile Engineers (SAE), the Association for the Advancement of Automotive Medicine (AAAM), the International Research Council on the Biomechanics of Impact (IRCOBI), as well as a variety of trauma-related biomedical journals.
               From the onset, the reader should understand that the purpose of examining injury thresholds is to aid in the design of safer vehicles, and it is to this end that all such criteria have been designed. No injury criterion has ever been developed with the intention of denying the likelihood of injury in an occupant after one has been diagnosed by a clinician, yet this is how it is currently used in the defense of crash injury litigation.
               The majority of crashes occur at relatively lower speed impacts, and as the speed change of collisions increases there are significantly fewer collisions. The majority of injuries also occur in relatively low speed crashes, although the most serious injuries do occur in higher speed crashes. The greatest proportion of the crash injury literature focuses on the minority of crashes that result in more severe to fatal injuries, and thus the literature is somewhat at odds with what the average clinician sees outside the emergency room on a regular basis.
               In the succeeding parts of this chapter that deal with the literature on injury thresholds, it will become readily apparent that these “thresholds” were not designed to be, not can they be used as a means of determining whether a specific  injury may have occurred in a specific case. As mentioned earlier, thresholds are derived from experimental rather than observational data, and are used to design and test vehicles for safety. The term or concept of “threshold” requires explanation anytime it is used. For example, it can refer to the level of frontal crash severity at which it is estimated there is less than an 80% risk of serious head injury, or it can refer to the speed change in a rear impact collision below which an injury to a crash test volunteer is highly likely to sustain injury. Without the specific parameters of the threshold, it is meaningless information.
               This is not to say that the use of experimental data does not allow for the assessment of relative injury risk prospectively for certain crashes. For example, it can be reasonably projected that, in a vehicle with a low head injury criterion (HIC) score in frontal crash test, there will be fewer severe head injuries than in a vehicle with a higher HIC for equivalent crash severities. However, it cannot be concluded that no serious head injuries will occur in the vehicle with the lowest HIC--regardless of collision speed--since the specific conditions of a crash and occupant cannot be predicted with 100% accuracy.
               The most egregious application of injury thresholds is as a means of denying the possibility of a medically-proven injury. Probability or risk of injury is only a tool for projecting a distribution of outcomes, and can never be used retrospectively to deny an outcome. After an injury has occurred in a crash and been medically validated, the “risk” if injury is 100% and it cannot be altered by prior outcomes. The appropriate use of probability can be compared with the game roulette. For a roulette wheel with 100 slots, it is reasonable to conclude that the pre-spin odds of the ball dropping into any particular slot are 1 in 100. After the ball drops into a particular slot the pre-spin odds are irrelevant to the outcome.

Thursday, November 8, 2012

Biomechanical Issues for Motor Vehicle Collisions


Biomechanical Issues for Motor Vehicle Collisions

It is important for the doctor to recognize that if a particular case goes into litigation, the insurance carrier will often hire a defense biomechanics expert, or medical expert that uses the conclusions of the biomechanists, to challenge the patient’s injuries and symptom logy, as well as the doctor’s diagnosis and treatment. The biomechanist can be an effective ally for the defense if he/she can attack the injury mechanisms and G forces (acceleration) generated during the collision, and influence a jury to believe the patient was not injured or simply had a trivial injury that would have resolved within a brief period of time with or without any treatment. These opinions are then typically reiterated by the defense medical expert, who is in agreement with the biomechanist, with the clear objective of downplaying the extent of the plaintiff’s injuries (bias) and attacking the treating doctors. In most cases the defense biomechanics expert will focus on the following issues:
               -Insufficient force to cause injury
               -Downplay the severity of injury, this giving the defense doctor basis to state that the treatment was excessive and unnecessary
               -Forces of collision are similar to those experienced in activities of daily living and therefore not responsible for post crash symptoms or injuries
               -Will often rely on limited living human volunteer testing (no subjects taken from general population) and extrapolate data to the specific patient
               -No injury mechanism
               -All post-traumatic symptoms were due to prior degenerative conditions or injuries and would have occurred at the same time frame absent the MVC

Wednesday, October 17, 2012

Inflammatory Outcomes


Inflammatory Outcomes
               
               Resolution is a possible outcome at this stage on condition that less than a critical number of cells have been destroyed. For more patients that come to our attention, this is an unlikely scenario.
               Suppuration, in the presence of infective microorganisms will result in pus formation. Pus consists of dead cell debris, living, dead and dying polymorphs suspended in the inflammatory exudate. Clearly the presence of an infection will delay the healing of a wound.
               Chronic inflammation does not necessarily imply inflammation of long duration, and may follow a transient or prolonged acute inflammatory stage. Essentially there are two forms of chronic inflammation: either the chronic reaction supervenes on the acute reaction or may in fact develop slowly with no initial acute phase. Chronic supervening on acute almost always involves some suppuration while chronic ab initio can have many causes including local irritants, poor circulation, some micro-organisms or immune disturbances. Chronic inflammation is usually more productive than exudative – it produces more fibrous material than inflammatory exudate. Frequently there is some tissue destruction, inflammation and attempted healing occurring simultaneously.
               Healing by fibrosis will most likely be taking place in the tissue repair scenario considered here. The fibrin deposits from the inflammatory stage will be partly removed by the fibrinolytic enzymes and will be gradually replaced by granulation tissue which becomes organized to form the scar tissue. Macrophages are largely responsible for the removal of the fibrin, allowing capillary budding and fibroblastic activity to proceed (proliferation). The greater the volume of damaged tissue, the greater the extent of, and the greater the density of, the resulting scar tissue. Chronic inflammation is usually accompanied by some fibrosis even in the absence of significant tissue destruction. The effects of acute inflammation are largely beneficial. The fluid exudate dilutes the toxins and escaped blood products include antibodies (and systemic drugs). The fibrinogen forms fibrin clots providing a mechanical barrier to the spread of micro-organisms (if present) and additionally assist phagocytosis. The gel-like consistency of the inflammatory exudate also makes a positive contribution by preventing the spread of the inflammatory mediators to surrounding, intact tissues.

Monday, September 17, 2012

Atlanta Population and Atlanta Demographics




Atlanta Population and Atlanta Demographics

Atlanta Population and Atlanta Demographics

Atlanta Population:
• 5,475,213 (2009 est.) in the 28-county Atlanta Metropolitan Statistical Area, designated by the Metro Atlanta Chamber of Commerce
• 4,124,300 in the 10-county Atlanta region
• 540,922 (2009 est.) in the City of Atlanta
Source: Atlanta Regional Commission, 2011; Census Bureau 2009
Ask any Atlantan to name a word to describe the changing Atlanta population, the word dynamic often comes to mind. It describes the rapid changes the city has seen and will see in Atlanta demographics over the next 10 years.

In the past six years, the metro area has added more than 458,568 people annually, more than any other in the United States. At this growth rate, the projected population of Atlanta's 20 core counties for 2020 is 6.4 million.

Atlanta has no natural boundaries, so the metropolitan area is large, encompassing 8,480.27 square miles. Across the region, 28 counties and 140 municipalities govern. The City of Atlanta, in the center of the metropolitan area, encompasses 131 square miles.

Growth means the Atlanta population has become more diverse. In fact, the city's racial diversity is greater than the nation as a whole. Atlanta is also younger than the U.S. population with an average age of 34 compared to 36 for the population as a whole. More than 35 percent of those who moved here from 2000 – 2004 came to Atlanta from a different state. The largest percentage of people came from New York and New Jersey.


Monday, September 10, 2012

Multiple-Vehicle Collisions


Multiple-Vehicle Collisions
               Multiple collisions, in which a vehicle is submitted to one or more successive collisions after the initial collision, are of special significance as far as the types of occupant injuries and the injury severity are concerned. One of the primary concerns for multiple impacts is the risk of the first impact resulting in the occupant being out-of-position (OOP) for the second crash. In addition, having injuries from a second impact superimposed upon injuries from the first impact may significantly alter the nature and extent of injuries, depending upon the circumstances. In some instances, the doctor will be confronted with a patient who has been involved in a multi-vehicle collision and has been requested to determine if the first or second crash was responsible for the injuries or the respective percentage of causation. The doctor’s opinion may be needed to determine apportionment for each collision. If two insurance carriers are involved, they may want to pass the blame for injuries to the other carrier. From a biomechanical perspective, multiple collisions are very challenging. Several issues that need to be considered:
               A) delta-V for first impact and the subsequent crash(s)
               B) mass ratios of the vehicles involved for each impact
               C) vehicle rotation for each crash, if occurring, and if so, how severe
               D) types of vehicles involved (i.e., SUV vs. a passenger car in a side impact). Height issues between the two vehicles and aggressive designs can result in differing injury patterns.
               E) intrusion extent into the occupant compartment for each vehicle, if any.
               F) contact point on the vehicle relative to the seating position of the occupant and proximity to the contact area, and whether the point of impact (POI) was rigid or soft. Rigid pillars and smaller structures will cause much higher acceleration levels than softer impacts or by larger objects capable of distributing loading over a larger area of the body.
               G) times of occupant acceleration. Longer impacts are better for occupants
               H) OOP issues for occupant at the time of each impact. Both impacts may have OOP issues.
               I) Second injury superimposed on first injury. Doctor needs to consider each impact with its own characteristics. For example, did each impact involve a head strike or did one impact cause inertial loading only? Need to consider the surface size and energy absorbing quality for each impact as well.
               J) Type and severity of first injury can make second impact more or less likely to cause other injuries.
               K) Patterns of injury related to vector of each crash.
               L) other human and vehicle factors.
              
               Temming and Zobel used a Volkswagen database of 1,620 occupants who suffered cervical spine injuries, concluding that about 23.5% of occupants in passenger cars were involved in at least one additional collision and 5.6% of occupants had a third impact following the initial crash. Another study found that multi-vehicle collisions occur in about 15% of total crashed, and the second collision speed has been found to be just as high as or higher than the first crash 43.2% of the time. An example is the rear-end collision in which one car is pushed into a vehicle ahead, and is struck on the side of the vehicle in the intersection, starting a chain reaction of traumatic events. Otte et al. concluded that injuries may be caused by almost all parts of the interior, due to actual impact situation and the consequent relative motion of the occupants.
               Fat et al. analyzed CCIS-UK data of 1,295 crashes looking for multiple impacts. Their study found that a secondary impact with another vehicle occurred in 22.6% of crashes and an additional 6.1% were involved in a third impact. The study concluded that there was a higher risk of occupants being seriously injured in multiple impacts, with the head having the highest frequency. The most common double impact events happened in frontal crashed followed by side impacts or side impacts followed by another side impact.

Thursday, August 23, 2012

Disc Injuries in Rear Impacts


Disc Injuries in Rear Impacts

               Discinjuries following rear-end impacts are often observed in clinical practice by physicians, many following in the low-speed delta-V range. I have seen many disc bulging or herniation cases over the years in the delta-V range of 4 to 10 mph. Most of these disc cases had preexisting degeneration in the spine noted with imaging studies or out-of-position issues leading to susceptibility. Smith evaluated 72 real-world rear impacts with accident reconstruction to determine the delta-Vs and the types of injuries that were diagnosed by physicians. Discbulges or herniations were found in the cervical spine with delta-Vs as low as 8km/h and in the lumbar spine as low as 11.3 km/h. Yoganandan et al. exposed four entire human cadavers to rear impacts with 4.4 to 6.8m/sec (9.8 to 15.2 mph) delta-V and a mean of 3.3 or 4.5g acceleration. After single impact, the cervical spine was analyzed for any traumatic findings using standard radiology and CT scanning. X-ray and CT analysis only found an avulsion fracture of the C5 body and C5-C6 disc distraction injury without a fracture. Then the specimens underwent cry sectioning to determine if any soft tissue injuries occurred that were not noted in the radiological evaluation. Cryosectioning revealed the following: annular tears at C5-C6, diastasis of C1-C2 and C5-C6 zygapophysical joints with associated tears of capsular ligaments, tears of C6-C7 ligamentum flavum, and tear/rupture of C5-C6 anterior longitudinal ligament.

Saturday, July 14, 2012

Chiropractic Adjustments for Newborns



Greater complications during delivery result in greater neurological insult to the newborn due to injury to the head and neck. Even after vaginal births, 4.6% of term neonates suffer unexplained brain bleeds and 10% suffer neonatal encephalopathy. Because so many children had been injured with forceps deliveries, (facial nerve palsy, tearing of cervical spine musculature) vacuum extraction was developed. Suction cups are placed on the newborn's head, and the baby is literally sucked out of the mother.

When utilized, 120 pounds of pressure goes through the baby's head and neck. Decapitation occurs at 140 pounds of pressure, to give you an idea of the high forces involved. Remember when you were a little girl or boy and there was a new baby you were being introduced to? Our parents always said, "Watch his head-you don't want to hurt him." We're careful because the fontanel's of the skull are so pliable, and the neck and brain are fragile and unprotected. This is why so many babies sustain injuries to their heads and neck during vacuum extraction-the force is far greater than their little bodies can tolerate.

Adjustments to newborns contain only ounces of force. But that force is directed into the spine to facilitate health and remove subluxations. We adjust babies as soon after birth as possible, to alleviate subluxations caused by in-utero constraint and the journey down through the birth canal. There has been a lot in the media lately about children not needing Chiropractic care, but there is no better way to get a head start in life. As you all know, Chiropractic care is not a cure for anything-it is a system of wellness to help us be who we're supposed to be. It is not a cure for ear infections, for colic, for allergies, for asthma, for frequent colds, nor for ADD/ADHD.

When we listen to mothers' stories of their pregnancy, labor and delivery, the children who suffer the most from the above complaints, are the ones who've had the greatest trouble with their births. Even relatively easy deliveries can result in subluxations. That's why every child should be checked, before problems with their health even develop. That's preventive care in the truest sense-preventing subluxations in mothers to prevent subluxations in their babies during childbirth. This is why every woman needs Chiropractic through pregnancy-so that the arrival of their baby is a wonderful experience. 

Wednesday, July 11, 2012

Is Chiropractic Safe During Pregnancy?


Not only is it safe to visit a chiropractor during your pregnancy, it’s also highly beneficial. All chiropractors are specially trained to treat pregnant women, but you may want to do a little research and find one who specializes in prenatal or perinatal care. Getting regularly adjusted while pregnant is a great way to relieve the added stress on your spine that comes along with the weight gain. It can also prevent sciatica, the inflammation of the sciatic nerve that runs from your lower back down through your legs and to your feet. It’s also important to maintain pelvic balance, which is oftentimes thrown off as your belly grows and your posture changes.

Besides making you feel better during pregnancy, getting regular chiropractic adjustments can also help control nausea, prevent a potential C-section, and has even been linked to reducing the amount of time some women spend in labor.  www.thebump.com

Chiropractic care through pregnancy is not only safe, it is essential. We can look at the implications of subluxation from a bio mechanical, hormonal and neurological standpoint. It is easy for all of us to see postural changes through pregnancy-the centre of gravity changes, the weight of the baby places increased pressure on the spine and pelvis, and towards the end of the pregnancy, changes are seen in gait pattern-the "waddle." What we can't see, are the millions of different hormonal changes and chemical reactions occurring both in the mother and the developing baby--all of which are controlled and coordinated through the nervous system.


Adjustments result in easier pregnancy, significantly decreased mean labor time, and assists new mothers back to prepartum health. In one study, women receiving Chiropractic care through their first pregnancy had 24% shorter labor times than the group not receiving Chiropractic, and multiparous subjects reported 39% shorter labor times. Thirty-nine percent-that's a massive difference. In addition, 84% of women report relief of back pain during pregnancy with Chiropractic care. Because the sacroiliac joints of the pelvis function better, there is significant less likelihood of back labor when receiving Chiropractic care through pregnancy.

Body position during delivery is also critical. Any late second stage labor position that denies postural sacral rotation denies the mother and the baby critical pelvic outlet diameter and jams the tip of the sacrum up to 4cm into the pelvic outlet. In other words, the popular semi-recumbent position places the laboring woman on her back onto the apex of the sacrum, which closes off the vital space needed for the baby to get through the pelvic outlet.

Friday, June 1, 2012

Overcoming Chiropractic Fear


If you have never been to a chiropractor before, it is only natural to have some apprehension on your first visit. If you are suffering from neck or back pain, visiting a chiropractor can alleviate and even eliminate pain, and yet some people are simply afraid to go. This is especially true if you don’t know what to expect. Knowledge is power and getting the facts and understanding what will happen can help you overcome your fear.

Book a Consultation

One of the best things you can do to overcome your fear of the chiropractor is to book a consultation appointment. Make sure when you book the appointment you let them know about your concerns. A good chiropractor will sit down with you and explain to you about chiropractic care, what it is and what will happen during your first adjustment. They will let you know that being adjusted does not cause any pain and in fact relieves it. They should be able to answer any questions you might have and may even have additional brochures or short movies for you to watch.

Ask Friends, Family and Co-Workers About Their Chiropractic Experiences

Another way to help alleviate your fear is to talk to others who have been treated by a chiropractor in the past or who are currently under chiropractic care. They will be able to tell you their stories of chiropractic success and let you know there is nothing to worry about. This is also a great way to get a referral and just knowing you are seeing a chiropractor that has been recommended can help.

Focus On Pain Relief

The most common reason people visit chiropractors is for back or neck pain relief. If this is you, try to stay focused on the pain relief. So many people continually suffer from neck and back pain that could so easily be alleviated and even eliminated through chiropractic care. Especially if you are seriously suffering, in the end, pain relief should outweigh fear.

Tuesday, May 29, 2012

Chiropractic Treatment of Knee Pain


Chiropractic treatment of knee pain

We have outlined the process of our treatment below to give you an idea of our approach. We would normally modify the treatment to suit each patient and their specific conditions. So the type of treatment that is most appropriate for one person is not necessarily right for another.
Stage 1: Reduce joint inflammation and reduce pain.
How?
  • Avoidance of aggravating factors, use of a support, sports tape
  • Ice to reduce inflammation, muscle spasm and pain
  • Soft tissue healing: laser, ultrasound and interferential therapy
Stage 2: Normalise joint function
How?
  • Specific chiropractic manipulation and mobilisation techniques to areas of restricted movement in the knee and surrounding joints, to increase movement, improve the function and reduce pain
  • Deep soft tissue massage, trigger-point therapy and cross-friction.
Stage 3: Rehabilitation exercise programme.
  • When appropriate we introduce exercises to improve strength, endurance and stability. We also work on improving your balance and your sense of joint position (proprioception). This will help you to return to normal and also prevent new injuries.

Friday, May 25, 2012

Symptoms of Poor Posture


Years and years of poor posture can trigger a series of symptoms in the average adult. They can start by:
Fatigue – you wake up and don’t feel like doing anything. Your muscles are weak, without energy, and so is your mind. Things lose brightness and taste. You think to yourself: “the solution to this problem would be to do some exercise”,  but ironically the problem is that you don’t feel like going out or doing exercise. A vicious cycle starts, which can only be ended by adopting a good posture.
Tight muscles in the neck, back, arms and legs. You’ll know that you’ve reached this stage because you’ll feel your muscles and ligaments stiff, tight and painful, specially neck and back muscles. That typical image of a person sitting in front of the computer and moving their head to relieve the pain says it all. Most of neck and back problems are the consequence of years of bad posture.
Joint stiffness and pain – this is especially dangerous in later years, because of the risk of arthritis, or what is called degenerative osteoarthritis. Joints are then very sensitive, since they’ve been supporting the weight of the body for years.

Friday, May 18, 2012

Disc Degeneration

Disc degeneration results when the cartilage that cushions the discs along the upper, middle and lower spine gets worn from overuse and the nerves become impinged or irritated. If the cartilage wears away completely, spinal discs can actually begin rubbing against one another, causing extreme pain. Often the supporting muscles get inflamed as well, further constricting the nerves and limiting blood flow to injured areas, worsening pain and suppressing the body's ability to heal. Effective treatment for disc degeneration includes spinal manipulation to relieve pain and inflammation, decompression, heat/ice therapies,  and stretching exercises to help alleviate spinal pressure.